Letter in response to glucagon-like peptide-1 mediates cardioprotection by remote ischaemic conditioning.

نویسندگان

  • Joel P Giblett
  • Stephen P Hoole
چکیده

TSP1–CD47 signaling is upregulated in clinical pulmonary hypertension and contributes to pulmonary arterial vasculopathy and dysfunction N. M. Rogers, M. Sharifi-Sanjani, M. Yao, K. Ghimire, R. Bienes-Martinez, S. M. Mutchler, H. E. Knupp, J. Baust, E. M. Novelli, M. Ross, C. St. Croix, J. C. Kutten, C. A. Czajka, J. C. Sembrat, M. Rojas, D. Labrousse-Arias, T. N. Bachman, R. R. Vanderpool, B. S. Zuckerbraun, H. C. Champion, A. L. Mora, A. C. Straub, R. A. Bilonick, M. J. Calzada, and J. S. Isenberg 15

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Glucagon-like peptide-1 (GLP-1) mediates cardioprotection by remote ischaemic conditioning

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Myocardial ischaemia-reperfusion injury can be significantly reduced by an episode(s) of ischaemia-reperfusion applied prior to or during myocardial ischaemia (MI) to peripheral tissue located at a distance from the heart; this phenomenon is called remote ischaemic conditioning (RIc). Here, we compared the efficacy of RIc in protecting the heart when the RIc stimulus is applied prior to, during...

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Ma Glucagon-like peptide-1-(7-36) amide (GLP-1) is a human incretin hormone responsible for the release of insulin in response to food. Pre-clinical and human physiological studies have demonstrated cardioprotection from ischemia-reperfusion injury. It can reduce infarct size, ischemic left ventricular dysfunction, and myocardial stunning. GLP-1 receptor agonists have also been shown to reduce ...

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Previous studies have shown that glucagon-like peptide-1 (GLP-1) provides cardiovascular benefits independent of its role on peripheral glycemic control. However, the precise mechanism(s) by which GLP-1 treatment renders cardioprotection during myocardial ischemia remain unresolved. Here we examined the role for GLP-1 treatment on glucose and fatty acid metabolism in normal and ischemic rat hea...

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عنوان ژورنال:
  • Cardiovascular research

دوره 113 1  شماره 

صفحات  -

تاریخ انتشار 2017